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Fig. 2 | Journal of Inflammation

Fig. 2

From: Lipid from electronic cigarette-aerosol both with and without nicotine induced pro-inflammatory macrophage polarization and disrupted phagocytosis

Fig. 2

ECIG-aerosol exposure induced lipid peroxidation and lipid accumulation. ECIG induced lipid peroxidation product malondialdehyde (MDA) (A) and MDA-modified proteins in MQ (B). Lipid scavenger receptors CD36 (C) and lipid accumulation were increased in MQ (D). Statistical significance (p ≤ 0.05) between Sham and exposure (nicotine or without nicotine) condition was indicated as *. Statistical significance between nicotine and without nicotine was indicated as #

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