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Fig. 1 | Journal of Inflammation

Fig. 1

From: Offspring sex affects the susceptibility to maternal smoking-induced lung inflammation and the effect of maternal antioxidant supplementation in mice

Fig. 1

Lung p-ERK1,2, p-JNK1,2, p-p38 and p- NF-κB in the offspring at P1. Protein levels of p-ERK1,2 a, b, p-JNK1,2 c, d, p-p38 e, f and p- NF-κB g, h in the lung of the male and female offspring at P1. Results are expressed as means ± SE, (male n = 8, female, n = 6–8). Data were analysed by one-way ANOVA followed by Tukey’s post hoc tests. *P < 0.05, **P < 0.01, *** P < 0.001, ****P < 0.0001. ERK, extracellular signal-regulated kinase; JNK, c-JUN N-terminal kinase; p38, p38 Mitogen-activated protein kinase; NF-κB: Nuclear factor-kB. SE, maternal smoke exposure; SE + LC, maternal smoke exposure with L-Carnitine supplement

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