Figure 2From: Upregulation of ICAM-1 in diabetic rats after transient forebrain ischemia and reperfusion injuryPischingert staining of the Nissl bodies in neurons of the frontal cortex after ischemia and reperfusion in normo- and hyperglycemic ischemic rats. (A,D) Normal Nissl substances were stained in the cytoplasm of sham control animals. (B,E) Decreased staining intensity observed in normoglycemic ischemia rats. (C,F) Significantly weaker staining of Nissl substances in hyperglycemic ischemic rats. (G) Bar graph indicating the mean optical density (OD) of the Nissl bodies in the three groups at different reperfusion times. (H) The number of degenerative neurons in the cerebral frontal cortex. Arrows indicate positive Pischingert staining of the Nissl bodies. *P < 0.05 vs. sham group, #P < 0.05 vs. normoglycemic group at an identical reperfusion stage. Magnification, 400X, bar = 20 μm.Back to article page